Mike Walden review author of acne no more program-It is a chronic
disease of the pilosebaceous unit characterized by the appearance on the trunk
and face a diverse rash combined with intense oily skin. Induced hyperactivity
of the sebaceous gland duct obstruction. The skin oils are trademarks in acne.
The sebaceous glands produce sebum that is a mixture of lipids
which after effect of microorganisms can be converted into free fatty acids
irritants causing acne lesions. The more severe form of acne, the greater is
the production of sebum.
The acne is not an infectious disease and is not spread from one
person to another. But both the skin surface and the pilosebaceous important
resource colonized by microorganisms such as propionic acid bacterium of acne,
epidermal aureus and pityrosporum oval of which the first is predominant. Thus
various treatments which reduce microorganisms improve the clinical picture.
The activity of the sebaceous glands under the control of androgen and highly
correlated with the levels of free testosterone.
Typical signs of acne is intense oiliness of the skin, the
comedones, the canary, the red papules and pustules, nodules, cysts, scars,
keloids and post inflammatory hyperpigmentation.
Factors that affect acne are:
Climatic conditions. The disease is worse in warm, humid and
tropical climatic conditions.
Drugs. Various formulations topically applied as a cosmetic oil or
other substances such as brilliantine applied to the scalp can cause clogging
pilosebaceous resources resulting in the appearance of comedones and akmoeidon
lesions.
Exclusion conditions of the skin. The tight fitting clothes cause
conditions exclusion of pilosebaceous resources resulting in people who wear
them to show akmoeides rash on thighs and buttocks. Also prolonged stay in
seats of plastic leads to sweating and over wetting of the skin resulting in
follicular obstruction and causing acne lesions.
Industrial causes. Industrial workers have akmoeideis damage when
exposed to halogenated hydrocarbons (chloracne).
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